Depression And Its Neurobiological
Beck, A. T. (2008). The evolution of the cognitive model of depression and its
neurobiological correlates. American Journal of Psychiatry, 165, 969977
Beck’s purpose for the book is to present findings from cognitive neuroscience and behavioral genetics with the accumulated research on the cognitive models. This opens new opportunities for integrated research on psychology. The author suggests that a comprehensive study of psychology and of biological correlates of depression can give us new understanding of the debilitating disorder. The authors attempt to integrate a different physiological puzzle of depression in which at the end product is a comprehensive cognitive model of depression. The findings of this model have raised the question of what are the factors that produce negative bias. Becker supports his answer through the clinical observation research which he has conducted. He further states that when patients are highly charged with dysfunctional attitudes beliefs about themselves. This hacks information processing which produces the negative bias leading to depression symptoms. According to him, the theory of consolidation of negative attitudes and negative self-concept along with associated motivational, effective and behavioral factors into the mode of depression are speculative. However it is useful as an explanatory construct. This is because the progression of process to depressive will lead to full blown depression that would be characterized by hyper activation of this conglomerate and diminution of realty test.
France, C. M., Lysaker, P. H., & Robinson, R. P (2007). The chemical imbalance explanation for depression: Origins, lay endorsement and clinical implications.
The authors discuss the origin, transmittals and accuracy of chemical imbalance explanation for depression. They present results from a group case study and discuss the medical implications with a short script of educating clients concerning depression’s chemical imbalance.The authors support their augment with studies using approximation and behavioral high risk design to corroborate the cognitive vulnerability hypothesis of depression. This is in support of their arguments that results from remitted studies on depression but have not achieved to do this. There suspicions are that the design features of previously conducted remitted designs were likely to prelude the findings from showing maladaptive cognitive patterns. Concerning the issue of the depressed individuals , their study of the participants showed more negative cognitive styles but not of greater levels of dysfunctional attitudes than individuals who had never been depressed.
Haeffel, G. J., Abramson, L. Y., Voelz, Z. R., Metalsky, G. I., Halberstadt, L., & Dykman, B. M.,. (2005). Negative cognitive styles, dysfunctional attitudes, and the remitted depression paradigm.
The authors conducted a study that features on the remitted design that has been successful in a biological vulnerability for depression studies. They have researched on the lifetime, prevalence of clinically significant depression, depressive symptoms and negative cognitive styles. The opinion of the authors is that this study of depression is not to reassure a design that is an optimal strategy but instead, there goal was to show that when a remitted depression study minimizes factors that can change cognitive vulnerability, then the study will obtain results that are consistent with those from behavioral high risk designs. The authors support their argument by the use of an optimal design called the CSQ and a much larger sample. The study is successful because it was able to capture the elusive cognitive vulnerability to depression factor on the depressives that are remitted. The authors assume that depressed individuals also qualify to be vulnerable to depression. This is opposed to the remitted design which states that if the maladaptive cognitive patterns provide vulnerability for depression. According to this logic cognitive pattern is not exhibited by previously depressed person which is not the actual case.
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